Upregulation of CX3CL1 mediated by NF-κB activation in dorsal root ganglion contributes to peripheral sensitization and chronic pain induced by oxaliplatin administration-Reference-Cited by-同舟云学术

Upregulation of CX3CL1 mediated by NF-κB activation in dorsal root ganglion contributes to peripheral sensitization and chronic pain induced by oxaliplatin administration

Published:2017-01 Issue: Volume:13 Page:174480691772625
ISSN:1744-8069
Container-title:Molecular Pain
language:en
Short-container-title:Mol Pain

Author:

Wang Jing¹,Zhang Xin-Sheng²,Tao Rong¹,Zhang Jie³,Liu Lin¹,Jiang Ying-Hai¹,Ma Song-He¹,Song Lin-Xia⁴,Xia Ling-Jie¹

Affiliation:

1. Department of Pain Management, Henan Provincial People's Hospital, Zhengzhou University, Zhengzhou, China

2. Department of Orthopaedics, Henan Provincial People's Hospital, Zhengzhou University, Zhengzhou, China

3. Department of Rehabilitation Medicine, Guangdong Woman and Children Hospital, Guangzhou, China

4. College of Life Science, Shandong University of Technology, Zibo, China

Publisher

SAGE Publications

Subject

Anesthesiology and Pain Medicine,Cellular and Molecular Neuroscience,Molecular Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1177/1744806917726256

Reference47 articles.

1. Oxaliplatin

2. Mechanisms Underlying Chemotherapy-Induced Neurotoxicity and the Potential for Neuroprotective Strategies

3. Persistent neuropathy after treatment with cisplatin and oxaliplatin

4. Effects of mitochondrial poisons on the neuropathic pain produced by the chemotherapeutic agents, paclitaxel and oxaliplatin

5. Prevention and treatment of chemotherapy-induced peripheral neuropathy

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