Synergistic activation of ERK1/2 between A-fiber neurons and glial cells in the DRG contributes to pain hypersensitivity after tissue injury-Reference-Cited by-同舟云学术

Synergistic activation of ERK1/2 between A-fiber neurons and glial cells in the DRG contributes to pain hypersensitivity after tissue injury

Published:2018-01 Issue: Volume:14 Page:174480691876750
ISSN:1744-8069
Container-title:Molecular Pain
language:en
Short-container-title:Mol Pain

Author:

Yamakita Shunsuke¹²,Horii Yasuhiko¹²,Takemura Hitomi¹²,Matsuoka Yutaka¹²,Yamashita Ayahiro¹²,Yamaguchi Yosuke¹²,Matsuda Megumi¹²,Sawa Teiji¹,Amaya Fumimasa¹²^ORCID

Affiliation:

1. Department of Anesthesiology, Kyoto Prefectural University of Medicine, Kyoto, Japan

2. Research Unit for the Neurobiology of Pain, Department of Anesthesiology, Kyoto Prefectural University of Medicine, Kyoto, Japan

Funder

Japan Society for the Promotion of Science

Publisher

SAGE Publications

Subject

Anesthesiology and Pain Medicine,Cellular and Molecular Neuroscience,Molecular Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1177/1744806918767508

Reference28 articles.

1. Pathophysiology of postoperative pain

2. Activation of p38 mitogen-activated protein kinase in the dorsal root ganglion contributes to pain hypersensitivity after plantar incision

3. Acquired Exchange Protein Directly Activated by Cyclic Adenosine Monophosphate Activity Induced by p38 Mitogen-activated Protein Kinase in Primary Afferent Neurons Contributes to Sustaining Postincisional Nociception

4. Molecular mechanisms of nociception

5. Different Mechanisms of Development and Maintenance of Experimental Incision-induced Hyperalgesia in Human Skin

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