P2X4-receptor participates in EAAT3 regulation via BDNF-TrkB signaling in a model of trigeminal allodynia-Reference-Cited by-同舟云学术

P2X4-receptor participates in EAAT3 regulation via BDNF-TrkB signaling in a model of trigeminal allodynia

Published:2018-01 Issue: Volume:14 Page:174480691879593
ISSN:1744-8069
Container-title:Molecular Pain
language:en
Short-container-title:Mol Pain

Author:

Liu Chaoyang¹,Zhang Yixin¹,Liu Qing¹,Jiang Li¹,Li Maolin¹,Wang Sha²,Long Ting¹,He Wei¹,Kong Xueying¹,Qin Guangcheng²,Chen Lixue²,Zhang Yuhong³,Zhou Jiying¹

Affiliation:

1. Department of Neurology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People’s Republic of China

2. Laboratory Research Center, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People’s Republic of China

3. The Clinical Molecular Diagnostics Center, The First Affiliated Hospital of Chongqing Medical University, Chongqing, People’s Republic of China

Funder

National Natural Science Foundation of China

Publisher

SAGE Publications

Subject

Anesthesiology and Pain Medicine,Cellular and Molecular Neuroscience,Molecular Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1177/1744806918795930

Reference50 articles.

1. P2X4 receptors induced in spinal microglia gate tactile allodynia after nerve injury

2. Region-specific disruption of the blood-brain barrier following repeated inflammatory dural stimulation in a rat model of chronic trigeminal allodynia

3. P2X4-Receptor-Mediated Synthesis and Release of Brain-Derived Neurotrophic Factor in Microglia Is Dependent on Calcium and p38-Mitogen-Activated Protein Kinase Activation

4. Up-Regulation of P2X4 Receptors in Spinal Microglia after Peripheral Nerve Injury Mediates BDNF Release and Neuropathic Pain

5. BDNF from microglia causes the shift in neuronal anion gradient underlying neuropathic pain

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