Blame it on the Inputs: Overexcited Entorhinal Inputs Drive Dentate Gyrus Hyperexcitability in a Mouse Model of Dravet Syndrome-Reference-Cited by-同舟云学术

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Blame it on the Inputs: Overexcited Entorhinal Inputs Drive Dentate Gyrus Hyperexcitability in a Mouse Model of Dravet Syndrome

Published:2022-09-23 Issue:6 Volume:22 Page:372-374
ISSN:1535-7597
Container-title:Epilepsy Currents
language:en
Short-container-title:Epilepsy Curr

Author:

Feng Yu¹,Shuman Tristan¹^ORCID

Affiliation:

1. Department of Neuroscience, Icahn School of Medicine at Mount Sinai, New York, NY, USA

Abstract

[Box: see text]

Funder

National Institute of Neurological Disorders and Stroke

Publisher

SAGE Publications

Subject

Neurology (clinical)

Link

http://journals.sagepub.com/doi/pdf/10.1177/15357597221112801

Reference10 articles.

1. Reduced sodium current in GABAergic interneurons in a mouse model of severe myoclonic epilepsy in infancy

2. Corticohippocampal circuit dysfunction in a mouse model of Dravet syndrome

3. De Novo Mutations in the Sodium-Channel Gene SCN1A Cause Severe Myoclonic Epilepsy of Infancy

4. Specific deletion of NaV1.1 sodium channels in inhibitory interneurons causes seizures and premature death in a mouse model of Dravet syndrome

5. A deleterious Nav1.1 mutation selectively impairs telencephalic inhibitory neurons derived from Dravet Syndrome patients

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1. Distinct changes to hippocampal and medial entorhinal circuits emerge across the progression of cognitive deficits in epilepsy;2024-03-14

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