Getting Excited About Chloride Cotransporters: Neuroinflammation and Inhibition
Author:
Affiliation:
1. Department of Neurology, University of Utah School of Medicine
2. Anticonvulsant Drug Development Program, Department of Pharmacology and Toxicology, College of Pharmacy, University of Utah
Abstract
Publisher
SAGE Publications
Link
https://journals.sagepub.com/doi/pdf/10.1177/15357597241238893
Reference9 articles.
1. A brain cytokine-independent switch in cortical activity marks the onset of sickness behavior triggered by acute peripheral inflammation
2. Acute neuroinflammation leads to disruption of neuronal chloride regulation and consequent hyperexcitability in the dentate gyrus
3. Role of the BDNF-TrkB pathway in KCC2 regulation and rehabilitation following neuronal injury: A mini review
4. Brain-Derived Neurotrophic Factor Inhibits the Function of Cation-Chloride Cotransporter in a Mouse Model of Viral Infection-Induced Epilepsy
5. Increased presynaptic excitability in a migraine with aura mutation
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