Idiopathic Intracranial Hypertension and Facial Palsy: Case Report and Review of the Literature

Author:

Tzoufi Meropi1,Makis Alexandros2,Grammeniatis Vasilios2,Nakou Iliada2,Exarchakos Georgios3,Asproudis Ioannis4,Zikou Anastasia5,Argyropoulou Maria5,Siamopoulou-Mavridou Antigone2

Affiliation:

1. Department of Child Health, the University of Ioannina Medical School, Ioannina, Greece,

2. Department of Child Health, the University of Ioannina Medical School, Ioannina, Greece

3. Department of Otorhinolaryngology, the University of Ioannina Medical School, Ioannina, Greece

4. Department of Ophthalmology the University of Ioannina Medical School, Ioannina, Greece

5. Department of Clinical Radiology, the University of Ioannina Medical School, Ioannina, Greece

Abstract

We present the case of an 11-year-old obese girl who presented with idiopathic intracranial hypertension affecting first the lateral abducens nerve. She received acetazolamide, but 5 days later she developed lateral, peripheral facial palsy. Imaging evaluation was normal, which primarily excluded cerebral venous thrombosis and sustained the initial diagnosis. Despite some complicating factors (obesity, elevated intracranial pressure), prednisolone was administered for a short-term period to counteract the facial palsy. Ophthalmological residuals resolved within almost 1.5 months, while facial palsy receded after 4 months. Peripheral facial palsy is an extremely rare, but not unknown condition in idiopathic intracranial hypertension. As a symptom, it should be investigated thoroughly, primarily to exclude cerebral venous sinus thrombosis, before it can be attributed to idiopathic intracranial hypertension. As far as treatment is concerned, corticosteroids can be added to the initial treatment with acetazolamide, without worsening already elevated intracranial hypertension or ophthalmologic findings.

Publisher

SAGE Publications

Subject

Clinical Neurology,Pediatrics, Perinatology, and Child Health

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