Angiotensin-(1–9) enhances stasis-induced venous thrombosis in the rat because of the impairment of fibrinolysis-Reference-Cited by-同舟云学术

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Angiotensin-(1–9) enhances stasis-induced venous thrombosis in the rat because of the impairment of fibrinolysis

Published:2013-07-24 Issue:1 Volume:15 Page:13-21
ISSN:1470-3203
Container-title:Journal of the Renin-Angiotensin-Aldosterone System
language:en
Short-container-title:J Renin Angiotensin Aldosterone Syst

Author:

Mogielnicki A¹²,Kramkowski K¹,Hermanowicz JM¹³,Leszczynska A¹,Przyborowski K¹,Buczko W⁴

Affiliation:

1. Department of Pharmacodynamics, Medical University of Bialystok, Poland

2. Department of Pharmacology and Toxicology, University of Varmia and Mazury, Poland

3. Department of Clinical Pharmacy, Medical University of Bialystok, Poland

4. Higher State Vocational School, Institute of Health Care, Poland

Publisher

Hindawi Limited

Subject

Endocrinology,Internal Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1177/1470320313498631

Reference28 articles.

1. Angiotensin II enhances thrombosis development in renovascular hypertensive rats

2. A Novel Angiotensin-Converting Enzyme–Related Carboxypeptidase (ACE2) Converts Angiotensin I to Angiotensin 1-9

3. A Human Homolog of Angiotensin-converting Enzyme

4. Inhibition of angiotensin-converting enzyme by des-Leu10-angiotensin I: a potential mechanism of endogenous angiotensin-converting enzyme regulation

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