Angiotensin II receptor blockade reduces salt sensitivity of blood pressure through restoration of renal nitric oxide synthesis in patients with diabetic nephropathy-Reference-Cited by-同舟云学术

Angiotensin II receptor blockade reduces salt sensitivity of blood pressure through restoration of renal nitric oxide synthesis in patients with diabetic nephropathy

Published:2012-08-02 Issue:1 Volume:14 Page:67-73
ISSN:1470-3203
Container-title:Journal of the Renin-Angiotensin-Aldosterone System
language:en
Short-container-title:J Renin Angiotensin Aldosterone Syst

Author:

Imanishi Masahito¹,Okada Noriyuki¹,Konishi Yoshio¹,Morikawa Takashi¹,Maeda Isseki¹,Kitabayashi Chizuko¹,Masada Masahiro²,Shirahashi Nobuo³,Wilcox Christopher S⁴,Nishiyama Akira⁵

Affiliation:

1. Department of Nephrology and Hypertension, Osaka City General Hospital, Japan

2. Laboratory of Biochemistry, Faculty of Horticulture, Chiba University, Japan

3. Department of Preventive Medicine and Environmental Health, Osaka City University Medical School, Japan

4. Division of Nephrology and Hypertension, Hypertension, Kidney and Vascular Health Center, Georgetown University, USA

5. Department of Pharmacology, and Hypertension and Kidney Research Center, Kagawa University Medical School, Japan

Publisher

Hindawi Limited

Subject

Endocrinology,Internal Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1177/1470320312454764

Reference27 articles.

1. Subtle Acquired Renal Injury as a Mechanism of Salt-Sensitive Hypertension

2. MECHANISMS MEDIATING PRESSURE NATRIURESIS: WHAT WE KNOW and WHAT WE NEED TO FIND OUT

3. The role of nitric oxide in hypertension and renal disease progression

4. Renal disease in rats with Type 2 diabetes is associated with decreased renal nitric oxide production

5. Role of abnormal nitric oxide systems in salt-sensitive hypertension

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