Evidence of changes in alpha-1/AT1 receptor function generated by diet-induced obesity-Reference-Cited by-同舟云学术

Evidence of changes in alpha-1/AT1 receptor function generated by diet-induced obesity

Published:2017-08-07 Issue:6 Volume:14 Page:485-493
ISSN:1479-1641
Container-title:Diabetes and Vascular Disease Research
language:en
Short-container-title:Diabetes and Vascular Disease Research

Author:

Juarez Esther¹,Tufiño Cecilia¹,Querejeta Enrique¹,Bracho-Valdes Ismael²,Bobadilla-Lugo Rosa A.¹

Affiliation:

1. Post-graduate studies and Investigation Department, Sección de Estudios de Posgrado e Investigación, Escuela Superior de Medicina, Instituto Politécnico Nacional, Mexico City, Mexico

2. Institute of Experimental and Clinical Therapeutics, Department of Physiology, Health Sciences University Center, University of Guadalajara, Guadalajara, Jalisco, Mexico

Publisher

SAGE Publications

Subject

Cardiology and Cardiovascular Medicine,Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1177/1479164117722069

Reference35 articles.

1. Diabetes Mellitus and Vascular Disease

2. The Renin-Angiotensin System and Its Receptors

3. Vascular Angiotensin Type 1 Receptor Expression Is Associated with Vascular Dysfunction, Oxidative Stress and Inflammation in Fructose-Fed Rats

4. Superoxide Mediates the Actions of Angiotensin II in the Central Nervous System

5. Angiotensin-(1-7) Augments Endothelium-dependent Relaxations of Porcine Coronary Arteries to Bradykinin by Inhibiting Angiotensin-Converting Enzyme 1

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. C21 preserves endothelial function in the thoracic aorta from DIO mice: role for AT2, Mas and B2 receptors;Clinical Science;2021-05

2. Differential Deleterious Impact of Highly Saturated Versus Monounsaturated Fat Intake on Vascular Function, Structure, and Mechanics in Mice;Nutrients;2021-03-19

3. AT1R antagonism improves metabolic and vascular changes of obesity-induced gestational diabetes mellitus;Pathophysiology;2019-06