Th1 to Th2 Cytokine Shifts in Nonobese Diabetic Mice: Sometimes an Outcome, Rather Than the Cause, of Diabetes Resistance Elicited by Immunostimulation-Reference-Cited by-同舟云学术

Th1 to Th2 Cytokine Shifts in Nonobese Diabetic Mice: Sometimes an Outcome, Rather Than the Cause, of Diabetes Resistance Elicited by Immunostimulation

Published:2001-01-15 Issue:2 Volume:166 Page:1352-1359
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Serreze David V.,Chapman Harold D.,Post Cristina M.,Johnson Ellis A.,Suarez-Pinzon Wilma L.,Rabinovitch Alex

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference44 articles.

1. Serreze, D. V.. 1998. The identity and ontogenic origins of autoreactive T lymphocytes in NOD mice. E. H. Leiter, and M. A. Atkinson, eds. NOD Mice and Related Strains: Research Applications in Diabetes, AIDS, Cancer, and Other Diseases 71 Landes Bioscience, Austin.

2. Immunoregulatory and Cytokine Imbalances in the Pathogenesis of IDDM: Therapeutic Intervention by Immunostimulation?

3. Insulin-Dependent Diabetes Mellitus

4. TECK: A Novel CC Chemokine Specifically Expressed by Thymic Dendritic Cells and Potentially Involved in T Cell Development

5. The effect of antigen dose on CD4+ T helper cell phenotype development in a T cell receptor-alpha beta-transgenic model.

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