Potentiation of Caspase-1 Activation by the P2X7 Receptor Is Dependent on TLR Signals and Requires NF-κB-Driven Protein Synthesis-Reference-Cited by-同舟云学术

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Potentiation of Caspase-1 Activation by the P2X7 Receptor Is Dependent on TLR Signals and Requires NF-κB-Driven Protein Synthesis

Published:2005-11-21 Issue:11 Volume:175 Page:7611-7622
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Kahlenberg J. Michelle,Lundberg Kathleen C.,Kertesy Sylvia B.,Qu Yan,Dubyak George R.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference62 articles.

1. Biologic basis for interleukin-1 in disease

2. Priming of Macrophages with Lipopolysaccharide Potentiates P2X7-mediated Cell Death via a Caspase-1-dependent Mechanism, Independently of Cytokine Production

3. Caspase-1-Deficient Mice Have Delayed Neutrophil Apoptosis and a Prolonged Inflammatory Response to Lipopolysaccharide-Induced Acute Lung Injury

4. ASC is essential for LPS-induced activation of procaspase-1 independently of TLR-associated signal adaptor molecules

5. Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf

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