Intracellular RIG-I Signaling Regulates TLR4-Independent Endothelial Inflammatory Responses to Endotoxin-Reference-Cited by-同舟云学术

Intracellular RIG-I Signaling Regulates TLR4-Independent Endothelial Inflammatory Responses to Endotoxin

Published:2016-04-29 Issue:11 Volume:196 Page:4681-4691
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Moser Jill^ORCID,Heeringa Peter^ORCID,Jongman Rianne M.,Zwiers Peter J.^ORCID,Niemarkt Anita E.,Yan Rui^ORCID,de Graaf Inge A.,Li Ranran,Ravasz Regan Erzsébet,Kümpers Philipp,Aird William C.,van Nieuw Amerongen Geerten P.,Zijlstra Jan G.^ORCID,Molema Grietje^ORCID,van Meurs Matijs

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference41 articles.

1. Effects of functional Toll-like receptor-4 mutations on the immune response to human and experimental sepsis

2. Lipopolysaccharide Clearance, Bacterial Clearance, and Systemic Inflammatory Responses Are Regulated by Cell Type–Specific Functions of TLR4 during Sepsis

3. Sepsis-induced organ failure is mediated by different pathways in the kidney and liver: Acute renal failure is dependent on MyD88 but not renal cell apoptosis

4. Role of Toll-Like Receptor 4 in Endotoxin-Induced Acute Renal Failure

5. Combination of Imipenem and TAK-242, a Toll-Like Receptor 4 Signal Transduction Inhibitor, Improves Survival in a Murine Model of Polymicrobial Sepsis

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