Characterization of Nitric Oxide Consumption Pathways by Normal, Chronic Granulomatous Disease and Myeloperoxidase-Deficient Human Neutrophils-Reference-Cited by-同舟云学术

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Characterization of Nitric Oxide Consumption Pathways by Normal, Chronic Granulomatous Disease and Myeloperoxidase-Deficient Human Neutrophils

Published:2002-11-15 Issue:10 Volume:169 Page:5889-5896
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Clark Stephen R.,Coffey Marcus J.,Maclean Rhona M.,Collins Peter W.,Lewis Malcolm J.,Cross Andrew R.,O’Donnell Valerie B.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference67 articles.

1. Nitric oxide: an endogenous modulator of leukocyte adhesion.

2. An endogenous inhibitor of nitric oxide synthase regulates endothelial adhesiveness for monocytes

3. Inhibition by nitric oxide-releasing compounds of E-selectin expression in and neutrophil adhesion to human endothelial cells

4. Inhibition by nitric oxide-donors of human polymorphonuclear leucocyte functions

5. Activated human neutrophils rapidly break down nitric oxide

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1. The Multiple Faces of Nitric Oxide in Chronic Granulomatous Disease: A Comprehensive Update;Biomedicines;2022-10-14

2. Oxidative responses and fungal infection biology;Seminars in Cell & Developmental Biology;2019-05

3. The discovery of nitro-fatty acids as products of metabolic and inflammatory reactions and mediators of adaptive cell signaling;Nitric Oxide;2018-07

4. Reactive Oxygen and Reactive Nitrogen Metabolites as Effector Molecules against Infectious Pathogens;The Innate Immune Response to Infection;2014-04-09

5. The role of leukotrienes in immunopathogenesis of rheumatoid arthritis;Modern Rheumatology;2014-03

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