Paralytic Autoimmune Myositis Develops in Nonobese Diabetic Mice Made Th1 Cytokine-Deficient by Expression of an IFN-γ Receptor β-Chain Transgene-Reference-Cited by-同舟云学术

Paralytic Autoimmune Myositis Develops in Nonobese Diabetic Mice Made Th1 Cytokine-Deficient by Expression of an IFN-γ Receptor β-Chain Transgene

Published:2003-03-01 Issue:5 Volume:170 Page:2742-2749
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Serreze David V.,Pierce Melissa A.,Post Cristina M.,Chapman Harold D.,Savage Holly,Bronson Roderick T.,Rothman Paul B.,Cox Gregory A.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference36 articles.

1. Serreze, D. V., E. H. Leiter. 2001. Genes and pathways underlying autoimmune diabetes in NOD mice. M. G. von Herrath, ed. Molecular Pathology of Insulin Dependent Diabetes Mellitus 31 Karger Press, New York.

2. Leiter, E. H.. 1990. The NOD mouse meets the “Nerup Hypothesis”: is diabetogenesis the result of a collection of common alleles present in unfavorable combinations?. P. Vardi, and E. Shafrir, eds. Frontiers in Diabetes Research: Lessons from Animal Diabetes III 54 Smith-Gordon, London.

3. Type I Diabetes and Multiple Sclerosis Patients Target Islet Plus Central Nervous System Autoantigens; Nonimmunized Nonobese Diabetic Mice Can Develop Autoimmune Encephalitis

4. Development of Spontaneous Autoimmune Peripheral Polyneuropathy in B7-2–Deficient Nod Mice

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