Arachidonic Acid Metabolism as a Potential Mediator of Cardiac Fibrosis Associated with Inflammation
Author:
Publisher
The American Association of Immunologists
Subject
Immunology,Immunology and Allergy
Reference82 articles.
1. Chronic Myocarditis Induced by T Cells Reactive to a Single Cardiac Myosin Peptide: Persistent Inflammation, Cardiac Dilatation, Myocardial Scarring and Continuous Myocyte Apoptosis
2. Myocardial Inflammatory Cell Infiltrates in Cases of Dilated Cardiomyopathy as a Determinant of Outcome Following Partial Left Ventriculectomy.
3. Anti-Monocyte Chemoattractant Protein-1 Gene Therapy Attenuates Left Ventricular Remodeling and Failure After Experimental Myocardial Infarction
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