IL-1α Is Essential for Oviduct Pathology during Genital Chlamydial Infection in Mice
Author:
Publisher
The American Association of Immunologists
Subject
Immunology,Immunology and Allergy
Reference62 articles.
1. Tubal factor infertility, with special regard to chlamydial salpingitis
2. Cervical Cytokines Associated With Chlamydia trachomatis Susceptibility and Protection
3. Pathology after Chlamydia trachomatis infection is driven by nonprotective immune cells that are distinct from protective populations
4. A role for CXC chemokine receptor-2 in the pathogenesis of urogenital Chlamydia muridarum infection in mice
5. Significant Role of IL-1 Signaling, but Limited Role of Inflammasome Activation, in Oviduct Pathology duringChlamydia muridarumGenital Infection
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1. Haematopoietic innate interleukin 17A production drives immunopathology in female mouse genital Chlamydia muridarum infection;Scandinavian Journal of Immunology;2024-01-21
2. Plasmid-mediated virulence in Chlamydia;Frontiers in Cellular and Infection Microbiology;2023-08-17
3. MAP4K4 promotes chlamydial infectivity and genital tract inflammatory injury by negatively regulating protective immunity in Chlamydia infection;2023-03-29
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5. Role of the human vaginal microbiota in the regulation of inflammation and sexually transmitted infection acquisition: Contribution of the non-human primate model to a better understanding?;Frontiers in Reproductive Health;2022-12-06
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