Inability of bm14 Mice to Respond to Theiler’s Murine Encephalomyelitis Virus Is Caused by Defective Antigen Presentation, Not Repertoire Selection-Reference-Cited by-同舟云学术

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Inability of bm14 Mice to Respond to Theiler’s Murine Encephalomyelitis Virus Is Caused by Defective Antigen Presentation, Not Repertoire Selection

Published:2005-02-22 Issue:5 Volume:174 Page:2756-2762
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Block Matthew S.,Mendez-Fernandez Yanice V.,Van Keulen Virginia P.,Hansen Michael J.,Allen Kathleen S.,Taboas Anya L.,Rodriguez Moses,Pease Larry R.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference38 articles.

1. The Origins of HLA-A,B,C Polymorphism

2. The Major Histocompatibility Complex of the Mouse

3. Spontaneous H-2 mutants provide evidence that a copy mechanism analogous to gene conversion generates polymorphism in the major histocompatibility complex.

4. The DNA sequence of the H-2kb gene: evidence for gene conversion as a mechanism for the generation of polymorphism in histocompatibilty antigens.

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Ovalbumin-specific CD4+ and CD8+ T cells contribute to different susceptibility for Theiler’s murine encephalomyelitis virus persistence;Frontiers in Immunology;2023-05-24

2. Theiler's Murine Encephalomyelitis Virus as a Vaccine Candidate for Immunotherapy;PLoS ONE;2011-05-20

3. Being a mouse in a man's world: what TMEV has taught us about human disease;Frontiers in Bioscience;2008

4. Hyperhomocysteinemic Alzheimer's mouse model of amyloidosis shows increased brain amyloid β peptide levels;Neurobiology of Disease;2006-06

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