Joint NOD2/RIPK2 Signaling Regulates IL-17 Axis and Contributes to the Development of Experimental Arthritis-Reference-Cited by-同舟云学术

Joint NOD2/RIPK2 Signaling Regulates IL-17 Axis and Contributes to the Development of Experimental Arthritis

Published:2012-04-09 Issue:10 Volume:188 Page:5116-5122
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Vieira Silvio M.,Cunha Thiago M.,França Rafael F. O.,Pinto Larissa G.,Talbot Jhimmy,Turato Walter M.,Lemos Henrique P.,Lima Jonilson B.,Verri Waldiceu A.,Almeida Sérgio C. L.,Ferreira Sergio H.,Louzada-Junior Paulo,Zamboni Dario S.,Cunha Fernando Q.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference39 articles.

1. Human Nod1 Confers Responsiveness to Bacterial Lipopolysaccharides

2. Nod2, a Nod1/Apaf-1 Family Member That Is Restricted to Monocytes and Activates NF-κB

3. NOD-LRR PROTEINS: Role in Host-Microbial Interactions and Inflammatory Disease

4. Nod2 Is a General Sensor of Peptidoglycan through Muramyl Dipeptide (MDP) Detection

5. An Induced Proximity Model for NF-κB Activation in the Nod1/RICK and RIP Signaling Pathways

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