APRIL Binding to BCMA Activates a JNK2–FOXO3–GADD45 Pathway and Induces a G2/M Cell Growth Arrest in Liver Cells-Reference-Cited by-同舟云学术

APRIL Binding to BCMA Activates a JNK2–FOXO3–GADD45 Pathway and Induces a G2/M Cell Growth Arrest in Liver Cells

Published:2012-10-15 Issue:10 Volume:189 Page:4748-4758
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Notas George,Alexaki Vassilia-Ismini,Kampa Marilena,Pelekanou Vassiliki,Charalampopoulos Ioannis,Sabour-Alaoui Sanaa,Pediaditakis Iosif,Dessirier Valérie,Gravanis Achille,Stathopoulos Efstathios N.,Tsapis Andreas,Castanas Elias

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference81 articles.

1. APRIL, a New Ligand of the Tumor Necrosis Factor Family, Stimulates Tumor Cell Growth

2. Biologically active APRIL is secreted following intracellular processing in the Golgi apparatus by furin convertase

3. BAFF, a Novel Ligand of the Tumor Necrosis Factor Family, Stimulates B Cell Growth

4. Interaction of the TNF homologues BLyS and APRIL with the TNF receptor homologues BCMA and TACI

5. Tumor Necrosis Factor (TNF) Receptor Superfamily Member TACI Is a High Affinity Receptor for TNF Family Members APRIL and BLyS

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