Interleukin-17 Acts Independently of TNF-α under Arthritic Conditions-Reference-Cited by-同舟云学术

Interleukin-17 Acts Independently of TNF-α under Arthritic Conditions

Published:2006-05-02 Issue:10 Volume:176 Page:6262-6269
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Koenders Marije I.,Lubberts Erik,van de Loo Fons A. J.,Oppers-Walgreen Birgitte,van den Bersselaar Liduine,Helsen Monique M.,Kolls Jay K.,Di Padova Franco E.,Joosten Leo A. B.,van den Berg Wim B.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference62 articles.

1. The combination of tumor necrosis factor ? blockade with interleukin-1 and interleukin-17 blockade is more effective for controlling synovial inflammation and bone resorption in an ex vivo model

2. Addition of interleukin 1 (IL1) and IL17 soluble receptors to a tumour necrosis factor alpha soluble receptor more effectively reduces the production of IL6 and macrophage inhibitory protein-3alpha and increases that of collagen in an in vitro model of rheumatoid synoviocyte activation

3. Human interleukin-17: A T cell-derived proinflammatory cytokine produced by the rheumatoid synovium

4. IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis

5. High Levels of IL-17 in Rheumatoid Arthritis Patients: IL-15 Triggers In Vitro IL-17 Production Via Cyclosporin A-Sensitive Mechanism

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