Selective Prostacyclin Receptor Agonism Augments Glucocorticoid-Induced Gene Expression in Human Bronchial Epithelial Cells-Reference-Cited by-同舟云学术

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Selective Prostacyclin Receptor Agonism Augments Glucocorticoid-Induced Gene Expression in Human Bronchial Epithelial Cells

Published:2009-10-30 Issue:10 Volume:183 Page:6788-6799
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Wilson Sylvia M.,Shen Pamela,Rider Christopher F.,Traves Suzanne L.,Proud David,Newton Robert,Giembycz Mark A.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference75 articles.

1. Pharmacodynamic profile of prostacyclin

2. Pharmacology and signaling of prostaglandin receptors: Multiple roles in inflammation and immune modulation

3. Characterization of the prostanoid receptor(s) on human blood monocytes at which prostaglandin E2inhibits lipopolysaccharide-induced tumour necrosis factor-αgeneration

4. A Key Role for Prostaglandin I2in Limiting Lung Mucosal Th2, But Not Th1, Responses to Inhaled Allergen

5. Prostaglandin I2analogs inhibit Th1 and Th2 effector cytokine production by CD4 T cells

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