Islet-Specific Expression of CXCL10 Causes Spontaneous Islet Infiltration and Accelerates Diabetes Development-Reference-Cited by-同舟云学术

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Islet-Specific Expression of CXCL10 Causes Spontaneous Islet Infiltration and Accelerates Diabetes Development

Published:2005-09-07 Issue:6 Volume:175 Page:3516-3524
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Rhode Antje,Pauza Mary E.,Barral Ana Maria,Rodrigo Evelyn,Oldstone Michael B. A.,von Herrath Matthias G.,Christen Urs

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference33 articles.

1. Among CXCR3 Chemokines, IFN-γ-Inducible Protein of 10 kDa (CXC Chemokine Ligand (CXCL) 10) but Not Monokine Induced by IFN-γ (CXCL9) Imprints a Pattern for the Subsequent Development of Autoimmune Disease

2. Neutralization of IFN-inducible protein 10/CXCL10 exacerbates experimental autoimmune encephalomyelitis

3. γ-Interferon transcriptionally regulates an early-response gene containing homology to platelet proteins

4. Biochemical characterization of a gamma interferon-inducible cytokine (IP-10).

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