Fine Mapping, Gene Content, Comparative Sequencing, and Expression Analyses Support Ctla4 and Nramp1 as Candidates for Idd5.1 and Idd5.2 in the Nonobese Diabetic Mouse-Reference-Cited by-同舟云学术

Fine Mapping, Gene Content, Comparative Sequencing, and Expression Analyses Support Ctla4 and Nramp1 as Candidates for Idd5.1 and Idd5.2 in the Nonobese Diabetic Mouse

Published:2004-06-21 Issue:1 Volume:173 Page:164-173
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Wicker Linda S.,Chamberlain Giselle,Hunter Kara,Rainbow Dan,Howlett Sarah,Tiffen Paul,Clark Jan,Gonzalez-Munoz Andrea,Cumiskey Anne Marie,Rosa Raymond L.,Howson Joanna M.,Smink Luc J.,Kingsnorth Amanda,Lyons Paul A.,Gregory Simon,Rogers Jane,Todd John A.,Peterson Laurence B.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference28 articles.

1. NOD Idd5 locus controls insulitis and diabetes and overlaps the orthologous CTLA4/IDDM12 and NRAMP1 loci in humans.

2. Further Mapping of the Idd5.1 Locus for Autoimmune Diabetes in NOD Mice

3. Natural resistance to infection with intracellular parasites: molecular genetics identifies Nramp1 as the Bcg/Ity/Lsh locus

4. Iron chelators modulate the fusogenic properties of Salmonella-containing phagosomes

5. Insulin Autoantibodies Are Associated With Islet Inflammation But Not Always Related to Diabetes Progression in NOD Congenic Mice

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