Cutting Edge: An In Vivo Requirement for STAT3 Signaling in TH17 Development and TH17-Dependent Autoimmunity-Reference-Cited by-同舟云学术

Cutting Edge: An In Vivo Requirement for STAT3 Signaling in TH17 Development and TH17-Dependent Autoimmunity

Published:2007-09-18 Issue:7 Volume:179 Page:4313-4317
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Harris Timothy J.,Grosso Joseph F.,Yen Hung-Rong,Xin Hong,Kortylewski Marcin,Albesiano Emilia,Hipkiss Edward L.,Getnet Derese,Goldberg Monica V.,Maris Charles H.,Housseau Franck,Yu Hua,Pardoll Drew M.,Drake Charles G.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference20 articles.

1. Rheumatoid Arthritis Synoviocyte Survival Is Dependent on Stat3

2. STAT expression and localization in the central nervous system during autoimmune encephalomyelitis in Lewis rats

3. pSTAT1, pSTAT3, and T-bet expression in peripheral blood mononuclear cells from relapsing-remitting multiple sclerosis patients correlates with disease activity

4. Sle1ab Mediates the Aberrant Activation of STAT3 and Ras-ERK Signaling Pathways in B Lymphocytes

5. Increased expression of STAT3 in SLE T cells contributes to enhanced chemokine-mediated cell migration

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