Secretion of IFN-γ but Not IL-17 by CD1d-Restricted NKT Cells Enhances Rejection of Skin Grafts Expressing Epithelial Cell-Derived Antigen-Reference-Cited by-同舟云学术

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Secretion of IFN-γ but Not IL-17 by CD1d-Restricted NKT Cells Enhances Rejection of Skin Grafts Expressing Epithelial Cell-Derived Antigen

Published:2010-04-21 Issue:10 Volume:184 Page:5663-5669
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Mattarollo Stephen R.,Yong Michelle,Tan Lieven,Frazer Ian H.,Leggatt Graham R.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference39 articles.

1. IL-22 is required for Th17 cell-mediated pathology in a mouse model of psoriasis-like skin inflammation.;Ma;J. Clin. Invest.,2008

2. Interleukin-17 in inflammatory skin disorders

3. Interleukin-22, a TH17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis

4. IL-17A directly inhibits TH1 cells and thereby suppresses development of intestinal inflammation

5. Cutting Edge: IFN-γ Regulates the Induction and Expansion of IL-17-Producing CD4 T Cells during Mycobacterial Infection

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