Acid Sphingomyelinase Is Required for Protection of Effector Memory T Cells against Glucocorticoid-Induced Cell Death-Reference-Cited by-同舟云学术

Acid Sphingomyelinase Is Required for Protection of Effector Memory T Cells against Glucocorticoid-Induced Cell Death

Published:2011-09-23 Issue:9 Volume:187 Page:4509-4516
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Tischner Denise,Theiss Jennifer,Karabinskaya Anna,van den Brandt Jens,Reichardt Sybille D.,Karow Ulrike,Herold Marco J.,Lühder Fred,Utermöhlen Olaf,Reichardt Holger M.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference40 articles.

1. Traditional Concepts and Future Avenues of Glucocorticoid Action in Experimental Autoimmune Encephalomyelitis and Multiple Sclerosis Therapy

2. Glucocorticoids in T cell apoptosis and function

3. BH3-only proteins Puma and Bim are rate-limiting for γ-radiation– and glucocorticoid-induced apoptosis of lymphoid cells in vivo

4. Resistance of Single-Positive Thymocytes to Glucocorticoid-Induced Apoptosis Is Mediated by CD28 Signaling

5. Glucocorticoids Engage Different Signal Transduction Pathways to Induce Apoptosis in Thymocytes and Mature T Cells

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