Human C3 Deficiency Associated with Impairments in Dendritic Cell Differentiation, Memory B Cells, and Regulatory T Cells-Reference-Cited by-同舟云学术

Human C3 Deficiency Associated with Impairments in Dendritic Cell Differentiation, Memory B Cells, and Regulatory T Cells

Published:2008-09-18 Issue:7 Volume:181 Page:5158-5166
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Ghannam Arije,Pernollet Martine,Fauquert Jean-Luc,Monnier Nicole,Ponard Denise,Villiers Marie-Bernadette,Péguet-Navarro Josette,Tridon Arlette,Lunardi Joel,Gerlier Denis,Drouet Christian

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference47 articles.

1. Human C′3: Evidence for the Liver as the Primary Site of Synthesis

2. Biosynthesis of the third component of complement (C3) in vitro by monocytes from both normal and homozygous C3-deficient humans.

3. Dendritic Cell Synthesis of C3 Is Required for Full T Cell Activation and Development of a Th1 Phenotype

4. Clinical Aspects and Molecular Basis of Primary Deficiencies of Complement Component C3 and its Regulatory Proteins Factor I and Factor H

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