Cutting Edge: Negative Regulation of Dendritic Cells through Acetylation of the Nonhistone Protein STAT-3-Reference-Cited by-同舟云学术

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Cutting Edge: Negative Regulation of Dendritic Cells through Acetylation of the Nonhistone Protein STAT-3

Published:2009-05-04 Issue:10 Volume:182 Page:5899-5903
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Sun Yaping,Chin Y. Eugene,Weisiger Elizabeth,Malter Chelsea,Tawara Isao,Toubai Tomomi,Gatza Erin,Mascagni Paolo,Dinarello Charles A.,Reddy Pavan

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference30 articles.

1. Histone deacetylase inhibition modulates indoleamine 2,3-dioxygenase–dependent DC functions and regulates experimental graft-versus-host disease in mice

2. Histone deacetylase inhibitor suberoylanilide hydroxamic acid reduces acute graft-versus-host disease and preserves graft-versus-leukemia effect

3. Reduction of graft-versus-host disease by histone deacetylase inhibitor suberonylanilide hydroxamic acid is associated with modulation of inflammatory cytokine milieu and involves inhibition of STAT1

4. Deacetylase inhibition promotes the generation and function of regulatory T cells

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