An Allelic Variant of Crry in the MurineSle1cLupus Susceptibility Interval Is Not Impaired in Its Ability To Regulate Complement Activation-Reference-Cited by-同舟云学术

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An Allelic Variant of Crry in the MurineSle1cLupus Susceptibility Interval Is Not Impaired in Its Ability To Regulate Complement Activation

Published:2010-07-21 Issue:4 Volume:185 Page:2331-2339
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Tchepeleva Svetlana N.,Thurman Joshua M.,Ruff Katherine,Perkins Stephen J.,Morel Laurence,Boackle Susan A.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference64 articles.

1. Systemic lupus erythematosus: an autoimmune disease of B cell hyperactivity

2. Susceptibility to lupus nephritis in the NZB/W model system

3. Polygenic control of susceptibility to murine systemic lupus erythematosus

4. Synovium as a source of increased amino-terminal parathyroid hormone-related protein expression in rheumatoid arthritis. A possible role for locally produced parathyroid hormone-related protein in the pathogenesis of rheumatoid arthritis.

5. Genetic dissection of systemic lupus erythematosus pathogenesis: Sle2 on murine chromosome 4 leads to B cell hyperactivity.;Mohan;J. Immunol.,1997

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1. Forward genetic approaches for elucidation of novel regulators of Lyme arthritis severity;Frontiers in Cellular and Infection Microbiology;2014-06-05

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