E1A Oncogene Enhancement of Caspase-2-Mediated Mitochondrial Injury Sensitizes Cells to Macrophage Nitric Oxide-Induced Apoptosis-Reference-Cited by-同舟云学术

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E1A Oncogene Enhancement of Caspase-2-Mediated Mitochondrial Injury Sensitizes Cells to Macrophage Nitric Oxide-Induced Apoptosis

Published:2008-06-03 Issue:12 Volume:180 Page:8272-8279
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Radke Jay R.,Siddiqui Zeba K.,Miura Tanya A.,Routes John M.,Cook James L.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference63 articles.

1. Adenovirus and cell cycle control

2. The protein products of the myc and myb oncogenes and adenovirus E1a are structurally related

3. Retinoblastoma family proteins as key targets of the small DNA virus oncoproteins

4. Adenovirus E1A gene-induced tumor rejection through cellular sensitization to immune and nonimmune apoptotic injuries

5. Use of Replicating Oncolytic Adenoviruses in Combination Therapy for Cancer

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1. Validation of the Intermolecular Disulfide Bond in Caspase-2;Biology;2024-01-17

2. Expression of Adenoviral E1A in Transformed Cells as an Additional Factor of HDACi-Dependent FoxO Regulation;Cells;2019-12-30

3. E1A oncogene induced sensitization to NK cell induced apoptosis requires PIDD and Caspase-2;Cell Death Discovery;2019-07-01

4. PIDD-dependent activation of caspase-2-mediated mitochondrial injury in E1A-induced cellular sensitivity to macrophage nitric oxide-induced apoptosis;Cell Death Discovery;2018-09-12

5. Expression of adenoviral E1A throws the PIDD switch;Cell Death & Disease;2017-01-05

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