Identification of Cellular Pathways of “Type 1,” Th17 T Cells, and TNF- and Inducible Nitric Oxide Synthase-Producing Dendritic Cells in Autoimmune Inflammation through Pharmacogenomic Study of Cyclosporine A in Psoriasis-Reference-Cited by-同舟云学术

Identification of Cellular Pathways of “Type 1,” Th17 T Cells, and TNF- and Inducible Nitric Oxide Synthase-Producing Dendritic Cells in Autoimmune Inflammation through Pharmacogenomic Study of Cyclosporine A in Psoriasis

Published:2008-01-21 Issue:3 Volume:180 Page:1913-1920
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Haider Asifa S.,Lowes Michelle A.,Suárez-Fariñas Mayte,Zaba Lisa C.,Cardinale Irma,Khatcherian Artemis,Novitskaya Inna,Wittkowski Knut M.,Krueger James G.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference46 articles.

1. Getting under the skin: the immunogenetics of psoriasis

2. Pathogenesis and therapy of psoriasis

3. Psoriasis vulgaris: cutaneous lymphoid tissue supports T-cell activation and ‘Type 1’ inflammatory gene expression

4. The Majority of Epidermal T Cells in Psoriasis Vulgaris Lesions can Produce Type 1 Cytokines, Interferon-γ, Interleukin-2, and Tumor Necrosis Factor-α, Defining TC1 (Cytotoxic T Lymphocyte) and TH1 Effector Populations:1 a Type 1 Differentiation Bias is also Measured in Circulating Blood T Cells in Psoriatic Patients

5. Identification of Common Transcriptional Regulatory Elements in Interleukin-17 Target Genes

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