Intracellular TLR Signaling: A Structural Perspective on Human Disease
Author:
Publisher
The American Association of Immunologists
Subject
Immunology,Immunology and Allergy
Reference50 articles.
1. The Toll–IL-1 receptor adaptor family grows to five members
2. Inhibition of Interleukin 1 Receptor/Toll-like Receptor Signaling through the Alternatively Spliced, Short Form of MyD88 Is Due to Its Failure to Recruit IRAK-4
3. A Common Dominant TLR5 Stop Codon Polymorphism Abolishes Flagellin Signaling and Is Associated with Susceptibility to Legionnaires' Disease
4. A stop codon polymorphism of Toll-like receptor 5 is associated with resistance to systemic lupus erythematosus
5. Distinct Mutations in IRAK-4 Confer Hyporesponsiveness to Lipopolysaccharide and Interleukin-1 in a Patient with Recurrent Bacterial Infections
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