IL-32: A Host Proinflammatory Factor against Influenza Viral Replication Is Upregulated by Aberrant Epigenetic Modifications during Influenza A Virus Infection-Reference-Cited by-同舟云学术

IL-32: A Host Proinflammatory Factor against Influenza Viral Replication Is Upregulated by Aberrant Epigenetic Modifications during Influenza A Virus Infection

Published:2010-10-01 Issue:9 Volume:185 Page:5056-5065
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Li Wei,Sun Wei,Liu Li,Yang Fang,Li Yongkui,Chen Yanni,Fang Jiali,Zhang Wenjing,Wu Jianguo,Zhu Ying

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference41 articles.

1. Activation of Interleukin-32 Pro-Inflammatory Pathway in Response to Influenza A Virus Infection

2. Increased level of IL-32 during human immunodeficiency virus infection suppresses HIV replication

3. Negative feedback regulation of IL-32 production by iNOS activation in response to dsRNA or influenza virus infection

4. Mycobacterium tuberculosis Induces Interleukin-32 Production through a Caspase- 1/IL-18/Interferon-γ-Dependent Mechanism

5. IL-32, a proinflammatory cytokine in rheumatoid arthritis

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