STAT3 Activation in Th17 and Th22 Cells Controls IL-22–Mediated Epithelial Host Defense during Infectious Colitis-Reference-Cited by-同舟云学术

STAT3 Activation in Th17 and Th22 Cells Controls IL-22–Mediated Epithelial Host Defense during Infectious Colitis

Published:2014-09-03 Issue:7 Volume:193 Page:3779-3791
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Backert Ingo,Koralov Sergei B.,Wirtz Stefan,Kitowski Vera,Billmeier Ulrike,Martini Eva,Hofmann Katharina,Hildner Kai,Wittkopf Nadine,Brecht Katrin,Waldner Maximilian,Rajewsky Klaus,Neurath Markus F.,Becker Christoph,Neufert Clemens

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference51 articles.

1. Control of pathogens and pathobionts by the gut microbiota

2. Muc2-Deficient Mice Spontaneously Develop Colitis, Indicating That MUC2 Is Critical for Colonic Protection

3. Regulation of Intestinal α-Defensin Activation by the Metalloproteinase Matrilysin in Innate Host Defense

4. Expression and regulation of antimicrobial peptides in the gastrointestinal tract

5. Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens

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