Increased Activation of Protein Kinase A Type I Contributes to the T Cell Deficiency in Common Variable Immunodeficiency
Author:
Affiliation:
1. *Section of Clinical Immunology and Infectious Diseases and Research Institute for Internal Medicine, Medical Department A, Rikshospitalet, Oslo, Norway; and
2. †Institute of Medical Biochemistry, University of Oslo, Oslo, Norway
Abstract
Publisher
The American Association of Immunologists
Subject
Immunology,Immunology and Allergy
Link
https://journals.aai.org/jimmunol/article-pdf/162/2/1178/1093072/im029901178o.pdf
Reference44 articles.
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2. Kammer, G. M.. 1988. The adenylate cyclase-cAMP-protein kinase A pathway and regulation of the immune response. Immunol. Today 9: 222
3. Skålhegg, B. S., B. F. Landmark, S. O. Døskeland, V. Hansson, T. Lea, T. Jahnsen. 1992. Cyclic AMP-dependent protein kinase type 1 mediate the inhibitory effects of 3′,5′-cyclic adenosine monophosphate on cell replication in human T lymphocytes. J. Biol. Chem. 256: 15707
4. Scott, J. D.. 1991. Cyclic nucleotide-dependent protein kinases. Pharmacol. Ther. 50: 123
5. Francis, S. H., J. D. Corbin. 1994. Structure and function of cyclic nucleotide-dependent protein kinases. Annu. Rev. Physiol. 56: 237
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