NLRP3 Regulates Neutrophil Functions and Contributes to Hepatic Ischemia–Reperfusion Injury Independently of Inflammasomes-Reference-Cited by-同舟云学术

NLRP3 Regulates Neutrophil Functions and Contributes to Hepatic Ischemia–Reperfusion Injury Independently of Inflammasomes

Published:2014-04-02 Issue:9 Volume:192 Page:4342-4351
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Inoue Yoshiyuki,Shirasuna Koumei,Kimura Hiroaki,Usui Fumitake,Kawashima Akira,Karasawa Tadayoshi,Tago Kenji,Dezaki Katsuya,Nishimura Satoshi,Sagara Junji,Noda Tetsuo,Iwakura Yoichiro,Tsutsui Hiroko,Taniguchi Shun’ichiro,Yanagisawa Ken,Yada Toshihiko,Yasuda Yoshikazu,Takahashi Masafumi

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference36 articles.

1. Mechanisms of Liver Injury. II. Mechanisms of neutrophil-induced liver cell injury during hepatic ischemia-reperfusion and other acute inflammatory conditions

2. Neutrophil Depletion and Chemokine Response after Liver Ischemia and Reperfusion

3. The Inflammasome NLRs in Immunity, Inflammation, and Associated Diseases

4. Role of the Inflammasome in Myocardial Infarction

5. Inflammasome Activation of Cardiac Fibroblasts Is Essential for Myocardial Ischemia/Reperfusion Injury

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