Cutting Edge: A Variant of the IL-23R Gene Associated with Inflammatory Bowel Disease Induces Loss of MicroRNA Regulation and Enhanced Protein Production-Reference-Cited by-同舟云学术

Cutting Edge: A Variant of the IL-23R Gene Associated with Inflammatory Bowel Disease Induces Loss of MicroRNA Regulation and Enhanced Protein Production

Published:2012-01-18 Issue:4 Volume:188 Page:1573-1577
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Zwiers Antonie,Kraal Laurens,van de Pouw Kraan Tineke C. T. M.,Wurdinger Thomas,Bouma Gerd,Kraal Georg

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference25 articles.

1. A Genome-Wide Association Study Identifies IL23R as an Inflammatory Bowel Disease Gene

2. Ulcerative colitis–risk loci on chromosomes 1p36 and 12q15 found by genome-wide association study

3. A Receptor for the Heterodimeric Cytokine IL-23 Is Composed of IL-12Rβ1 and a Novel Cytokine Receptor Subunit, IL-23R

4. Novel p19 Protein Engages IL-12p40 to Form a Cytokine, IL-23, with Biological Activities Similar as Well as Distinct from IL-12

5. The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17–producing effector T helper cells in vivo

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