Cutting Edge: CD4 T Cells Reactive to an Islet Amyloid Polypeptide Peptide Accumulate in the Pancreas and Contribute to Disease Pathogenesis in Nonobese Diabetic Mice-Reference-Cited by-同舟云学术

Cutting Edge: CD4 T Cells Reactive to an Islet Amyloid Polypeptide Peptide Accumulate in the Pancreas and Contribute to Disease Pathogenesis in Nonobese Diabetic Mice

Published:2013-09-16 Issue:8 Volume:191 Page:3990-3994
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Baker Rocky L.,Delong Thomas,Barbour Gene,Bradley Brenda,Nakayama Maki,Haskins Kathryn

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference23 articles.

1. Effector Function of Diabetogenic CD4 Th1 T Cell Clones: A Central Role for TNF-α

2. Pathogenic T‐Cell Clones in Autoimmune Diabetes: More Lessons from the NOD Mouse

3. Chromogranin A is an autoantigen in type 1 diabetes

4. Islet Amyloid Polypeptide Is a Target Antigen for Diabetogenic CD4+ T Cells

5. MHC Class II Tetramers

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