Vγ4 γδ T Cell-Derived IL-17A Negatively Regulates NKT Cell Function in Con A-Induced Fulminant Hepatitis-Reference-Cited by-同舟云学术

Vγ4 γδ T Cell-Derived IL-17A Negatively Regulates NKT Cell Function in Con A-Induced Fulminant Hepatitis

Published:2011-10-10 Issue:10 Volume:187 Page:5007-5014
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Zhao Na,Hao Jianlei,Ni Yuanyuan,Luo Wei,Liang Ruifang,Cao Guangchao,Zhao Yapu,Wang Puyue,Zhao Liqing,Tian Zhigang,Flavell Richard,Hong Zhangyong,Han Jihong,Yao Zhi,Wu Zhenzhou,Yin Zhinan

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference44 articles.

1. A T cell-dependent experimental liver injury in mice inducible by concanavalin A.

2. Critical contribution of liver natural killer T cells to a murine model of hepatitis

3. IFN-γ/STAT1 acts as a proinflammatory signal in T cell-mediated hepatitis via induction of multiple chemokines and adhesion molecules: a critical role of IRF-1

4. Augmentation of Vα14 Nkt Cell–Mediated Cytotoxicity by Interleukin 4 in an Autocrine Mechanism Resulting in the Development of Concanavalin a–Induced Hepatitis

5. Interferon gamma plays a critical role in T cell-dependent liver injury in mice initiated by concanavalin A

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