Cutting Edge: Caspase-1 Independent IL-1β Production Is Critical for Host Resistance toMycobacterium tuberculosisand Does Not Require TLR Signaling In Vivo-Reference-Cited by-同舟云学术

Cutting Edge: Caspase-1 Independent IL-1β Production Is Critical for Host Resistance toMycobacterium tuberculosisand Does Not Require TLR Signaling In Vivo

Published:2010-03-03 Issue:7 Volume:184 Page:3326-3330
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Mayer-Barber Katrin D.,Barber Daniel L.,Shenderov Kevin,White Sandra D.,Wilson Mark S.,Cheever Allen,Kugler David,Hieny Sara,Caspar Patricia,Núñez Gabriel,Schlueter Dirk,Flavell Richard A.,Sutterwala Fayyaz S.,Sher Alan

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference26 articles.

1. Immunity to Tuberculosis

2. Fatal Mycobacterium tuberculosis infection despite adaptive immune response in the absence of MyD88

3. MyD88-Deficient Mice Display a Profound Loss in Resistance to Mycobacterium tuberculosis Associated with Partially Impaired Th1 Cytokine and Nitric Oxide Synthase 2 Expression

4. TLR9 regulates Th1 responses and cooperates with TLR2 in mediating optimal resistance to Mycobacterium tuberculosis

5. Containment of aerogenicMycobacterium tuberculosis infection in mice does not require MyD88 adaptor function for TLR2, -4 and -9

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