Cooperation Between CD44 and LFA-1/CD11a Adhesion Receptors in Lymphokine-Activated Killer Cell Cytotoxicity
Author:
Affiliation:
1. †Department of Oral Biochemistry, Kanagawa Dental College, Kanagawa, Japan
2. *Amgen Institute, and Ontario Cancer Institute, Department of Medical Biophysics and Immunology, University of Toronto, Toronto, Ontario, Canada; and
Abstract
Publisher
The American Association of Immunologists
Subject
Immunology,Immunology and Allergy
Link
https://journals.aai.org/jimmunol/article-pdf/160/12/5781/1085274/im129805781o.pdf
Reference43 articles.
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3. Bancroft, G. J.. 1993. The role of natural killer cells in innate resistance to infection. Curr. Opin. Immunol. 5: 503
4. Das, B., M. O. Mondragon, S.-Z. Tao, A. J. Norin. 1997. Preferential interaction of a novel tumor surface protein (p38.5) with natural killer cells. J. Exp. Med. 185: 1735
5. Grimm, E. A., R. J. Robb, J. A. Roth, L. M. Neckers, L. B. Rachman, D. J. Wilson, S. A. Rosenberg. 1983. Lymphokine-activated killer cell phenomenon. III. Evidence that IL-2 is sufficient for direct activation of peripheral lymphocytes into lymphokine-activated killer cells. J. Exp. Med. 158: 1356
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