Cutting Edge: Mast Cells Express IL-17A in Rheumatoid Arthritis Synovium-Reference-Cited by-同舟云学术

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Cutting Edge: Mast Cells Express IL-17A in Rheumatoid Arthritis Synovium

Published:2010-03-03 Issue:7 Volume:184 Page:3336-3340
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Hueber Axel J.,Asquith Darren L.,Miller Ashley M.,Reilly Jim,Kerr Shauna,Leipe Jan,Melendez Alirio J.,McInnes Iain B.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference25 articles.

1. Interferon-γ regulates susceptibility to collagen-induced arthritis through suppression of interleukin-17

2. Human interleukin-17: A T cell-derived proinflammatory cytokine produced by the rheumatoid synovium

3. In vivo activated monocytes from the site of inflammation in humans specifically promote Th17 responses

4. Monocyte Derived Interleukin (IL)-23 Is an Important Determinant of Synovial IL-17A Expression in Rheumatoid Arthritis

5. IL-17 and Th17 Cells

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2. N-demethylsinomenine metabolite and its prototype sinomenine activate mast cells via MRGPRX2 and aggravate anaphylaxis;Frontiers in Pharmacology;2024-07-31

3. Rheumatoid arthritis-associated complications during pregnancy and its effect on offspring: comprehensive review;Inflammopharmacology;2024-04-30

4. Impact of TNF and IL-33 Cytokines on Mast Cells in Neuroinflammation;International Journal of Molecular Sciences;2024-03-13

5. Mast cells express IL17A, IL17F and RORC, are activated and persist with IL-17 production in resolved skin of patients with chronic plaque-type psoriasis;2024-02-16

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