Small Molecule Mimetics of α-Helical Domain of IRAK2 Attenuate the Proinflammatory Effects of IL-33 in Asthma-like Mouse Models
Author:
Publisher
The American Association of Immunologists
Subject
Immunology,Immunology and Allergy
Reference42 articles.
1. IL-33, an Interleukin-1-like Cytokine that Signals via the IL-1 Receptor-Related Protein ST2 and Induces T Helper Type 2-Associated Cytokines
2. A putative protein of a growth specific cDNA from BALB/C-3T3 cells is highly similar to the extracellular portion of mouse interleukin 1 receptor
3. Presence and Expression of a Novel Variant Form of ST2 Gene Product in Human Leukemic Cell Line UT-7/GM
4. Soluble ST2 Blocks Interleukin-33 Signaling in Allergic Airway Inflammation
5. A Large-Scale, Consortium-Based Genomewide Association Study of Asthma
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1. Strategies targeting IL-33/ST2 axis in the treatment of allergic diseases;Biochemical Pharmacology;2023-12
2. IRAK2 Has a Critical Role in Promoting Feed-Forward Amplification of Epidermal Inflammatory Responses;Journal of Investigative Dermatology;2021-10
3. ILC2 Proliferated by IL-33 Stimulation Alleviates Acute Colitis in Rag1-/- Mouse through Promoting M2 Macrophage Polarization;Journal of Immunology Research;2020-06-25
4. Endogenous IL-33 and Its Autoamplification of IL-33/ST2 Pathway Play an Important Role in Asthma;The Journal of Immunology;2020-01-27
5. miR‐497a‐5p attenuates lipopolysaccharide‐induced inflammatory injury by targeting IRAK2;Journal of Cellular Physiology;2019-05-30
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