Islet-Specific Expression of IL-10 Promotes Diabetes in Nonobese Diabetic Mice Independent of Fas, Perforin, TNF Receptor-1, and TNF Receptor-2 Molecules-Reference-Cited by-同舟云学术

Islet-Specific Expression of IL-10 Promotes Diabetes in Nonobese Diabetic Mice Independent of Fas, Perforin, TNF Receptor-1, and TNF Receptor-2 Molecules

Published:2000-09-01 Issue:5 Volume:165 Page:2841-2849
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Balasa Balaji,Van Gunst Kurt,Jung Nadja,Balakrishna Deepika,Santamaria Pere,Hanafusa Toshiaki,Itoh Naoto,Sarvetnick Nora

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference30 articles.

1. Insulin-Dependent Diabetes Mellitus

2. Genetic Control of Autoimmune Diabetes in the Nod Mouse

3. The Nonobese Diabetic Mouse as a Model of Autoimmune Diabetes: Immune Dysregulation Gets the NOD

4. IL-10 is necessary and sufficient for autoimmune diabetes in conjunction with NOD MHC homozygosity.

5. Transgenic expression of IL-10 in pancreatic islet A cells accelerates autoimmune insulitis and diabetes in non-obese diabetic mice

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