Cutting Edge: RIP1 Kinase Activity Is Dispensable for Normal Development but Is a Key Regulator of Inflammation in SHARPIN-Deficient Mice-Reference-Cited by-同舟云学术

Cutting Edge: RIP1 Kinase Activity Is Dispensable for Normal Development but Is a Key Regulator of Inflammation in SHARPIN-Deficient Mice

Published:2014-05-12 Issue:12 Volume:192 Page:5476-5480
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Berger Scott B.,Kasparcova Viera,Hoffman Sandy,Swift Barb,Dare Lauren,Schaeffer Michelle,Capriotti Carol,Cook Michael,Finger Joshua,Hughes-Earle Angela,Harris Philip A.,Kaiser William J.,Mocarski Edward S.,Bertin John,Gough Peter J.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference17 articles.

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5. Identification of RIP1 kinase as a specific cellular target of necrostatins

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