Uracil DNA Glycosylase Disruption Blocks Ig Gene Conversion and Induces Transition Mutations-Reference-Cited by-同舟云学术

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Uracil DNA Glycosylase Disruption Blocks Ig Gene Conversion and Induces Transition Mutations

Published:2005-12-19 Issue:1 Volume:176 Page:365-371
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Saribasak Huseyin,Saribasak Nesibe Nur,Ipek Fatih M.,Ellwart Joachim W.,Arakawa Hiroshi,Buerstedde Jean-Marie

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference15 articles.

1. Class Switch Recombination and Hypermutation Require Activation-Induced Cytidine Deaminase (AID), a Potential RNA Editing Enzyme

2. Activation-Induced Cytidine Deaminase (AID) Deficiency Causes the Autosomal Recessive Form of the Hyper-IgM Syndrome (HIGM2)

3. Requirement of the Activation-Induced Deaminase ( AID ) Gene for Immunoglobulin Gene Conversion

4. AID Is Essential for Immunoglobulin V Gene Conversion in a Cultured B Cell Line

5. Immunoglobulin Isotype Switching Is Inhibited and Somatic Hypermutation Perturbed in UNG-Deficient Mice

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1. Unfolding the symbiosis of AID, chromatin remodelers, and epigenetics–The ACE phenomenon of antibody diversity;Immunology Letters;2024-10

2. Molecular Mechanism of Activation-Induced Cytidine Deaminase;Molecular Biology of B Cells;2024

3. Cell Cycle–Mediated Regulation of Secondary Ig Diversification;The Journal of Immunology;2023-03-31

4. Deciphering Genetic Alterations of Hairy Cell Leukemia and Hairy Cell Leukemia-like Disorders in 98 Patients;Cancers;2022-04-10

5. RNA-directed DNA repair and antibody somatic hypermutation;Trends in Genetics;2021-11

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