Cyclin-Dependent Kinase 4 Hyperactivity Promotes Autoreactivity in the Immune System but Protects Pancreatic β Cell Mass from Autoimmune Destruction in the Nonobese Diabetic Mouse Model-Reference-Cited by-同舟云学术

Cyclin-Dependent Kinase 4 Hyperactivity Promotes Autoreactivity in the Immune System but Protects Pancreatic β Cell Mass from Autoimmune Destruction in the Nonobese Diabetic Mouse Model

Published:2008-01-04 Issue:2 Volume:180 Page:1189-1198
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Marzo Nuria,Ortega Sagrario,Stratmann Thomas,García Ainhoa,Ríos Martín,Giménez América,Gomis Ramon,Mora Conchi

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference49 articles.

1. Adult pancreatic β-cells are formed by self-duplication rather than stem-cell differentiation

2. Beta-cell proliferation and apoptosis in the developing normal human pancreas and in hyperinsulinism of infancy.

3. Dynamics of β-cell Mass in the Growing Rat Pancreas: Estimation With a Simple Mathematical Model

4. Very Slow Turnover of β-Cells in Aged Adult Mice

5. β-Cell Deficit and Increased β-Cell Apoptosis in Humans With Type 2 Diabetes

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