Bacteroides fragilisEnterotoxin Upregulates Intercellular Adhesion Molecule-1 in Endothelial Cells via an Aldose Reductase-, MAPK-, and NF-κB–Dependent Pathway, Leading to Monocyte Adhesion to Endothelial Cells-Reference-Cited by-同舟云学术

Bacteroides fragilisEnterotoxin Upregulates Intercellular Adhesion Molecule-1 in Endothelial Cells via an Aldose Reductase-, MAPK-, and NF-κB–Dependent Pathway, Leading to Monocyte Adhesion to Endothelial Cells

Published:2011-07-01 Issue:4 Volume:187 Page:1931-1941
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Roh Hyun Cheol,Yoo Do Young,Ko Su Hyuk,Kim Young-Jeon,Kim Jung Mogg

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference50 articles.

1. Bacteroides fragilis Enterotoxin Gene Sequences in Patients with Inflammatory Bowel Disease

2. Association of EnterotoxigenicBacteroides fragilisInfection with Inflammatory Diarrhea

3. Enterotoxigenic Bacteroides fragilis : a Rogue among Symbiotes

4. A human colonic commensal promotes colon tumorigenesis via activation of T helper type 17 T cell responses

5. Mitogen-activated protein kinase and activator protein-1 dependent signals are essential forBacteroides fragilis enterotoxin-induced enteritis

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4. Bacteroides fragilis Enterotoxin Induces Sulfiredoxin-1 Expression in Intestinal Epithelial Cell Lines Through a Mitogen-Activated Protein Kinases- and Nrf2-Dependent Pathway, Leading to the Suppression of Apoptosis;International Journal of Molecular Sciences;2020-07-29

5. Bacteroides fragilis enterotoxin upregulates heme oxygenase-1 in dendritic cells via reactive oxygen species-, mitogen-activated protein kinase-, and Nrf2-dependent pathway;World Journal of Gastroenterology;2020-01-21