Elevated Mitochondrial Reactive Oxygen Species Generation Affects the Immune Response via Hypoxia-Inducible Factor-1α in Long-LivedMclk1+/−Mouse Mutants-Reference-Cited by-同舟云学术

Elevated Mitochondrial Reactive Oxygen Species Generation Affects the Immune Response via Hypoxia-Inducible Factor-1α in Long-LivedMclk1+/−Mouse Mutants

Published:2009-12-09 Issue:2 Volume:184 Page:582-590
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Wang Dantong,Malo Danielle,Hekimi Siegfried

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference46 articles.

1. Redistribution of Intracellular Oxygen in Hypoxia by Nitric Oxide: Effect on HIF1α

2. Oxygen sensing requires mitochondrial ROS but not oxidative phosphorylation

3. Mitochondrial complex III is required for hypoxia-induced ROS production and cellular oxygen sensing

4. Mitochondrial dysfunction resulting from loss of cytochrome c impairs cellular oxygen sensing and hypoxic HIF-α activation

5. The Qo site of the mitochondrial complex III is required for the transduction of hypoxic signaling via reactive oxygen species production

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