Compensatory Mechanisms Allow Undersized Anchor-Deficient Class I MHC Ligands To Mediate Pathogenic Autoreactive T Cell Responses-Reference-Cited by-同舟云学术

Compensatory Mechanisms Allow Undersized Anchor-Deficient Class I MHC Ligands To Mediate Pathogenic Autoreactive T Cell Responses

Published:2014-07-25 Issue:5 Volume:193 Page:2135-2146
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Lamont Deanna,Mukherjee Gayatri,Kumar P. Rajesh,Samanta Dibyendu,McPhee Caroline G.,Kay Thomas W. H.,Almo Steven C.,DiLorenzo Teresa P.,Serreze David V.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference40 articles.

1. Major Histocompatibility Complex Class I-Deficient NOD-B2mnull Mice are Diabetes and Insulitis Resistant

2. Genetic Disassociation of Autoimmunity and Resistance to Costimulation Blockade-Induced Transplantation Tolerance in Nonobese Diabetic Mice

3. Insulitis in human type 1 diabetes

4. Individual Nonobese Diabetic Mice Exhibit Unique Patterns of CD8+ T Cell Reactivity to Three Islet Antigens, Including the Newly Identified Widely Expressed Dystrophia Myotonica Kinase

5. Spontaneous Autoimmune Diabetes in Monoclonal T Cell Nonobese Diabetic Mice

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